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Showing 1 to 12 of 319 entries
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Werner Syndrome.

Journal of biomedicine & biotechnology

Chen L, Oshima J.
PMID: 12488583
J Biomed Biotechnol. 2002;2(2):46-54. doi: 10.1155/S1110724302201011.

Werner syndrome is a premature aging disease caused by the mutation in the WRN gene. The cloning and characterization of the WRN gene and its product allows investigators to study the disease and the human aging process at molecular...

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Chen L, Oshima J. Werner Syndrome. J Biomed Biotechnol. 2002;2(2):46-54doi: 10.1155/S1110724302201011.
Chen, L., & Oshima, J. (2002). Werner Syndrome. Journal of biomedicine & biotechnology, 2(2), 46-54. https://doi.org/10.1155/S1110724302201011
Chen, Lishan, and Oshima, Junko. "Werner Syndrome." Journal of biomedicine & biotechnology vol. 2,2 (2002): 46-54. doi: https://doi.org/10.1155/S1110724302201011
Chen L, Oshima J. Werner Syndrome. J Biomed Biotechnol. 2002;2(2):46-54. doi: 10.1155/S1110724302201011. PMID: 12488583; PMCID: PMC153784.
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Recombinational DNA Repair in Cancer and Normal Cells: The Challenge of Functional Analysis.

Journal of biomedicine & biotechnology

Willers H, Xia F, Powell SN.
PMID: 12488588
J Biomed Biotechnol. 2002;2(2):86-93. doi: 10.1155/S1110724302204027.

A major goal of current cancer research is to understand the functional consequences of mutations in recombinational DNA repair genes. The introduction of artificial recombination substrates into living cells has evolved into a powerful tool to perform functional analysis...

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Willers H, Xia F, Powell SN. Recombinational DNA Repair in Cancer and Normal Cells: The Challenge of Functional Analysis. J Biomed Biotechnol. 2002;2(2):86-93doi: 10.1155/S1110724302204027.
Willers, H., Xia, F., & Powell, S. N. (2002). Recombinational DNA Repair in Cancer and Normal Cells: The Challenge of Functional Analysis. Journal of biomedicine & biotechnology, 2(2), 86-93. https://doi.org/10.1155/S1110724302204027
Willers, Henning, et al. "Recombinational DNA Repair in Cancer and Normal Cells: The Challenge of Functional Analysis." Journal of biomedicine & biotechnology vol. 2,2 (2002): 86-93. doi: https://doi.org/10.1155/S1110724302204027
Willers H, Xia F, Powell SN. Recombinational DNA Repair in Cancer and Normal Cells: The Challenge of Functional Analysis. J Biomed Biotechnol. 2002;2(2):86-93. doi: 10.1155/S1110724302204027. PMID: 12488588; PMCID: PMC153788.
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Two Novel Frame Shift, Recurrent and De Novo Mutations in the ITGB2 (CD18) Gene Causing Leukocyte Adhesion Deficiency in a Highly Inbred North African Population.

Journal of biomedicine & biotechnology

Fathallah DM, Jamal T, Barbouche MR, Bejaoui M, Hariz MB, Dellagi K.
PMID: 12488604
J Biomed Biotechnol. 2001;1(3):114-121. doi: 10.1155/S1110724301000250.

We have identified four different mutations causing leukocyte adhesion Deficiency (LAD) in the ITGB2 gene of patients from a highly inbred population. Two were novel single-bp deletions (1497delG and 1920delG) causing frame shift and the two others were the...

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Fathallah DM, Jamal T, Barbouche MR, et al. Two Novel Frame Shift, Recurrent and De Novo Mutations in the ITGB2 (CD18) Gene Causing Leukocyte Adhesion Deficiency in a Highly Inbred North African Population. J Biomed Biotechnol. 2001;1(3):114-121doi: 10.1155/S1110724301000250.
Fathallah, D. M., Jamal, T., Barbouche, M. R., Bejaoui, M., Hariz, M. B., & Dellagi, K. (2001). Two Novel Frame Shift, Recurrent and De Novo Mutations in the ITGB2 (CD18) Gene Causing Leukocyte Adhesion Deficiency in a Highly Inbred North African Population. Journal of biomedicine & biotechnology, 1(3), 114-121. https://doi.org/10.1155/S1110724301000250
Fathallah, D. M., et al. "Two Novel Frame Shift, Recurrent and De Novo Mutations in the ITGB2 (CD18) Gene Causing Leukocyte Adhesion Deficiency in a Highly Inbred North African Population." Journal of biomedicine & biotechnology vol. 1,3 (2001): 114-121. doi: https://doi.org/10.1155/S1110724301000250
Fathallah DM, Jamal T, Barbouche MR, Bejaoui M, Hariz MB, Dellagi K. Two Novel Frame Shift, Recurrent and De Novo Mutations in the ITGB2 (CD18) Gene Causing Leukocyte Adhesion Deficiency in a Highly Inbred North African Population. J Biomed Biotechnol. 2001;1(3):114-121. doi: 10.1155/S1110724301000250. PMID: 12488604; PMCID: PMC129056.
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Induction of Apoptosis in Rat Peripheral Blood Lymphocytes by the Anticancer Drug CI-994 (Acetyldinaline)(*).

Journal of biomedicine & biotechnology

Graziano MJ, Spoon TA, Cockrell EA, Rowse PE, Gonzales AJ.
PMID: 12488610
J Biomed Biotechnol. 2001;1(2):52-61. doi: 10.1155/S1110724301000146.

CI-994 (acetyldinaline) is an investigational anticancer drug currently in clinical trials. In preclinical safety studies in rats and dogs, CI-994 resulted in significant toxicity to bone marrow and lymphoid tissue. To determine if apoptosis was involved in CI-994 toxicity,...

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Graziano MJ, Spoon TA, Cockrell EA, et al. Induction of Apoptosis in Rat Peripheral Blood Lymphocytes by the Anticancer Drug CI-994 (Acetyldinaline)(*). J Biomed Biotechnol. 2001;1(2):52-61doi: 10.1155/S1110724301000146.
Graziano, M. J., Spoon, T. A., Cockrell, E. A., Rowse, P. E., & Gonzales, A. J. (2001). Induction of Apoptosis in Rat Peripheral Blood Lymphocytes by the Anticancer Drug CI-994 (Acetyldinaline)(*). Journal of biomedicine & biotechnology, 1(2), 52-61. https://doi.org/10.1155/S1110724301000146
Graziano, Michael J., et al. "Induction of Apoptosis in Rat Peripheral Blood Lymphocytes by the Anticancer Drug CI-994 (Acetyldinaline)(*)." Journal of biomedicine & biotechnology vol. 1,2 (2001): 52-61. doi: https://doi.org/10.1155/S1110724301000146
Graziano MJ, Spoon TA, Cockrell EA, Rowse PE, Gonzales AJ. Induction of Apoptosis in Rat Peripheral Blood Lymphocytes by the Anticancer Drug CI-994 (Acetyldinaline)(*). J Biomed Biotechnol. 2001;1(2):52-61. doi: 10.1155/S1110724301000146. PMID: 12488610; PMCID: PMC113775.
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The Thermal Stability of the Fusarium solani pisi Cutinase as a Function of pH.

Journal of biomedicine & biotechnology

Petersen SB, Fojan P, Petersen EI, Petersen MT.
PMID: 12488611
J Biomed Biotechnol. 2001;1(2):62-69. doi: 10.1155/S1110724301000249.

We have investigated the thermal stability of the Fusarium solani pisi cutinase as a function of pH, in the range from pH 2-12. Its highest enzymatic activity coincides with the pH-range at which it displays its highest thermal stability....

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Petersen SB, Fojan P, Petersen EI, et al. The Thermal Stability of the Fusarium solani pisi Cutinase as a Function of pH. J Biomed Biotechnol. 2001;1(2):62-69doi: 10.1155/S1110724301000249.
Petersen, S. B., Fojan, P., Petersen, E. I., & Petersen, M. T. (2001). The Thermal Stability of the Fusarium solani pisi Cutinase as a Function of pH. Journal of biomedicine & biotechnology, 1(2), 62-69. https://doi.org/10.1155/S1110724301000249
Petersen, Steffen B., et al. "The Thermal Stability of the Fusarium solani pisi Cutinase as a Function of pH." Journal of biomedicine & biotechnology vol. 1,2 (2001): 62-69. doi: https://doi.org/10.1155/S1110724301000249
Petersen SB, Fojan P, Petersen EI, Petersen MT. The Thermal Stability of the Fusarium solani pisi Cutinase as a Function of pH. J Biomed Biotechnol. 2001;1(2):62-69. doi: 10.1155/S1110724301000249. PMID: 12488611; PMCID: PMC113781.
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Vectorial Transcellular Calcium Transport in Intestine: Integration of Current Models.

Journal of biomedicine & biotechnology

Larsson D, Nemere I.
PMID: 12488574
J Biomed Biotechnol. 2002;2(3):117-119. doi: 10.1155/S1110724302205045.

In spite of decades of research, the exact subcellular pathway for calcium transport in intestine has not been elucidated. In this mini-review, we present three models for vectorial movement of calcium across the cell: facilitated (cytoplasmic) diffusion, vesicular/lysosomal transport,...

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Larsson D, Nemere I. Vectorial Transcellular Calcium Transport in Intestine: Integration of Current Models. J Biomed Biotechnol. 2002;2(3):117-119doi: 10.1155/S1110724302205045.
Larsson, D., & Nemere, I. (2002). Vectorial Transcellular Calcium Transport in Intestine: Integration of Current Models. Journal of biomedicine & biotechnology, 2(3), 117-119. https://doi.org/10.1155/S1110724302205045
Larsson, Dennis, and Nemere, Ilka. "Vectorial Transcellular Calcium Transport in Intestine: Integration of Current Models." Journal of biomedicine & biotechnology vol. 2,3 (2002): 117-119. doi: https://doi.org/10.1155/S1110724302205045
Larsson D, Nemere I. Vectorial Transcellular Calcium Transport in Intestine: Integration of Current Models. J Biomed Biotechnol. 2002;2(3):117-119. doi: 10.1155/S1110724302205045. PMID: 12488574; PMCID: PMC161364.
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Perillyl Alcohol Inhibits Breast Cell Migration without Affecting Cell Adhesion.

Journal of biomedicine & biotechnology

Wagner JE, Huff JL, Rust WL, Kingsley K, Plopper GE.
PMID: 12488578
J Biomed Biotechnol. 2002;2(3):136-140. doi: 10.1155/S1110724302207020.

The monoterpene d-limonene exhibits chemotherapeutic and chemopreventive potential in breast cancer patients. D-limonene and its related compounds, perillyl alcohol and perillyl aldehyde, were chosen as candidate drugs for application in a screen for nontoxic inhibitors of cell migration. Using...

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Wagner JE, Huff JL, Rust WL, et al. Perillyl Alcohol Inhibits Breast Cell Migration without Affecting Cell Adhesion. J Biomed Biotechnol. 2002;2(3):136-140doi: 10.1155/S1110724302207020.
Wagner, J. E., Huff, J. L., Rust, W. L., Kingsley, K., & Plopper, G. E. (2002). Perillyl Alcohol Inhibits Breast Cell Migration without Affecting Cell Adhesion. Journal of biomedicine & biotechnology, 2(3), 136-140. https://doi.org/10.1155/S1110724302207020
Wagner, Johanna E., et al. "Perillyl Alcohol Inhibits Breast Cell Migration without Affecting Cell Adhesion." Journal of biomedicine & biotechnology vol. 2,3 (2002): 136-140. doi: https://doi.org/10.1155/S1110724302207020
Wagner JE, Huff JL, Rust WL, Kingsley K, Plopper GE. Perillyl Alcohol Inhibits Breast Cell Migration without Affecting Cell Adhesion. J Biomed Biotechnol. 2002;2(3):136-140. doi: 10.1155/S1110724302207020. PMID: 12488578; PMCID: PMC161368.
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Quantitative Trait Loci Associated with Foliar Trigonelline Accumulation in Glycine Max L.

Journal of biomedicine & biotechnology

Cho Y, Njiti VN, Chen X, Triwatayakorn K, Kassem MA, Meksem K, Lightfoot DA, Wood AJ.
PMID: 12488580
J Biomed Biotechnol. 2002;2(3):151-157. doi: 10.1155/S1110724302204039.

The objective of this study was to utilize a Glycine max RIL population to (1) evaluate foliar trigonelline (TRG) content in field-grown soybean, (2) determine the heritability of TRG accumulation, and (3) identify DNA markers linked to quantitative trait...

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Cho Y, Njiti VN, Chen X, et al. Quantitative Trait Loci Associated with Foliar Trigonelline Accumulation in Glycine Max L. J Biomed Biotechnol. 2002;2(3):151-157doi: 10.1155/S1110724302204039.
Cho, Y., Njiti, V. N., Chen, X., Triwatayakorn, K., Kassem, M. A., Meksem, K., Lightfoot, D. A., & Wood, A. J. (2002). Quantitative Trait Loci Associated with Foliar Trigonelline Accumulation in Glycine Max L. Journal of biomedicine & biotechnology, 2(3), 151-157. https://doi.org/10.1155/S1110724302204039
Cho, Youngkoo, et al. "Quantitative Trait Loci Associated with Foliar Trigonelline Accumulation in Glycine Max L." Journal of biomedicine & biotechnology vol. 2,3 (2002): 151-157. doi: https://doi.org/10.1155/S1110724302204039
Cho Y, Njiti VN, Chen X, Triwatayakorn K, Kassem MA, Meksem K, Lightfoot DA, Wood AJ. Quantitative Trait Loci Associated with Foliar Trigonelline Accumulation in Glycine Max L. J Biomed Biotechnol. 2002;2(3):151-157. doi: 10.1155/S1110724302204039. PMID: 12488580; PMCID: PMC161363.
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Different Forms of Vanadate on Sugar Transport in Insulin Target and Nontarget Cells.

Journal of biomedicine & biotechnology

Germinario RJ, Colby-Germinario SP, Posner BI, Nahm K.
PMID: 12488596
J Biomed Biotechnol. 2002;2(1):22-30. doi: 10.1155/S1110724302000402.

The effects of several vanadates (ie, orthovanadate, pervanadate, and two stable peroxovanadium compounds) on basal and insulin-stimulated 2-DG transport in insulin target and nontarget cell lines are reported, herein. In nontarget cells, exposure to vanadates (5 x 10(-6) to...

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Germinario RJ, Colby-Germinario SP, Posner BI, et al. Different Forms of Vanadate on Sugar Transport in Insulin Target and Nontarget Cells. J Biomed Biotechnol. 2002;2(1):22-30doi: 10.1155/S1110724302000402.
Germinario, R. J., Colby-Germinario, S. P., Posner, B. I., & Nahm, K. (2002). Different Forms of Vanadate on Sugar Transport in Insulin Target and Nontarget Cells. Journal of biomedicine & biotechnology, 2(1), 22-30. https://doi.org/10.1155/S1110724302000402
Germinario, Ralph J., et al. "Different Forms of Vanadate on Sugar Transport in Insulin Target and Nontarget Cells." Journal of biomedicine & biotechnology vol. 2,1 (2002): 22-30. doi: https://doi.org/10.1155/S1110724302000402
Germinario RJ, Colby-Germinario SP, Posner BI, Nahm K. Different Forms of Vanadate on Sugar Transport in Insulin Target and Nontarget Cells. J Biomed Biotechnol. 2002;2(1):22-30. doi: 10.1155/S1110724302000402. PMID: 12488596; PMCID: PMC139119.
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High-Pressure Biotechnology in Medicine and Pharmaceutical Science.

Journal of biomedicine & biotechnology

Masson P, Tonello C, Balny C.
PMID: 12488614
J Biomed Biotechnol. 2001;1(2):85-88. doi: 10.1155/S1110724301000158.

High-pressure (HP) biotechnology is an emerging technique initially applied for food processing and more recently in pharmaceutical and medical sciences. Pressure can stabilize enzymes and modulate both their activity and specificity. HP engineering of proteins may be used for...

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Masson P, Tonello C, Balny C. High-Pressure Biotechnology in Medicine and Pharmaceutical Science. J Biomed Biotechnol. 2001;1(2):85-88doi: 10.1155/S1110724301000158.
Masson, P., Tonello, C., & Balny, C. (2001). High-Pressure Biotechnology in Medicine and Pharmaceutical Science. Journal of biomedicine & biotechnology, 1(2), 85-88. https://doi.org/10.1155/S1110724301000158
Masson, Patrick, et al. "High-Pressure Biotechnology in Medicine and Pharmaceutical Science." Journal of biomedicine & biotechnology vol. 1,2 (2001): 85-88. doi: https://doi.org/10.1155/S1110724301000158
Masson P, Tonello C, Balny C. High-Pressure Biotechnology in Medicine and Pharmaceutical Science. J Biomed Biotechnol. 2001;1(2):85-88. doi: 10.1155/S1110724301000158. PMID: 12488614; PMCID: PMC113776.
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Baculoviruses as Vectors for Gene Therapy against Human Prostate Cancer.

Journal of biomedicine & biotechnology

Stanbridge LJ, Dussupt V, Maitland NJ.
PMID: 12721513
J Biomed Biotechnol. 2003;2003(2):79-91. doi: 10.1155/S1110724303209049.

Current curative strategies for prostate cancer are restricted to the primary tumour, and the effect of treatments to control metastatic disease is not sustained. Therefore, the application of gene therapy to prostate cancer is an attractive alternative. Baculoviruses are...

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Stanbridge LJ, Dussupt V, Maitland NJ. Baculoviruses as Vectors for Gene Therapy against Human Prostate Cancer. J Biomed Biotechnol. 2003;2003(2):79-91doi: 10.1155/S1110724303209049.
Stanbridge, L. J., Dussupt, V., & Maitland, N. J. (2003). Baculoviruses as Vectors for Gene Therapy against Human Prostate Cancer. Journal of biomedicine & biotechnology, 2003(2), 79-91. https://doi.org/10.1155/S1110724303209049
Stanbridge, Lindsay J., et al. "Baculoviruses as Vectors for Gene Therapy against Human Prostate Cancer." Journal of biomedicine & biotechnology vol. 2003,2 (2003): 79-91. doi: https://doi.org/10.1155/S1110724303209049
Stanbridge LJ, Dussupt V, Maitland NJ. Baculoviruses as Vectors for Gene Therapy against Human Prostate Cancer. J Biomed Biotechnol. 2003;2003(2):79-91. doi: 10.1155/S1110724303209049. PMID: 12721513; PMCID: PMC323953.
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Targeting Radiotherapy to Cancer by Gene Transfer.

Journal of biomedicine & biotechnology

Mairs RJ, Boyd M.
PMID: 12721515
J Biomed Biotechnol. 2003;2003(2):102-109. doi: 10.1155/S1110724303209062.

Targeted radionuclide therapy is an alternative method of radiation treatment which uses a tumor-seeking agent carrying a radioactive atom to deposits of tumor, wherever in the body they may be located. Recent experimental data signifies promise for the amalgamation...

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Mairs RJ, Boyd M. Targeting Radiotherapy to Cancer by Gene Transfer. J Biomed Biotechnol. 2003;2003(2):102-109doi: 10.1155/S1110724303209062.
Mairs, R. J., & Boyd, M. (2003). Targeting Radiotherapy to Cancer by Gene Transfer. Journal of biomedicine & biotechnology, 2003(2), 102-109. https://doi.org/10.1155/S1110724303209062
Mairs, R. J., and Boyd, M.. "Targeting Radiotherapy to Cancer by Gene Transfer." Journal of biomedicine & biotechnology vol. 2003,2 (2003): 102-109. doi: https://doi.org/10.1155/S1110724303209062
Mairs RJ, Boyd M. Targeting Radiotherapy to Cancer by Gene Transfer. J Biomed Biotechnol. 2003;2003(2):102-109. doi: 10.1155/S1110724303209062. PMID: 12721515; PMCID: PMC323955.
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Showing 1 to 12 of 319 entries